rabbit α gpr83 Search Results


rabbit α gpr83  (Alomone Labs)
91
Alomone Labs rabbit α gpr83
a, b, In aged mice, ablation of <t>Gpr83</t> + neurons by injection of PEN-SAP reverses mechanical hyperknesis ( a ; control blank SAP, n = 7; PEN-SAP, n =6) and prevents spontaneous scratching ( b ). c, d, Ablation of Tacr1 + neurons in aged mice by injection of SSP-SAP has no effect on mechanical itch sensitivity ( c ; SAP, n = 7; SSP-SAP, n =6) or spontaneous scratching ( d ). e, f, Mechanical itch sensitivity ( e ) and spontaneous scratching ( f ) are increased by administration of CNO to P56 NPY::Cre; Lbx1 FlpO ; R26 ds-hM4D mice (hM4D + ; n = 7) as compared to control NPY::Cre; R26 ds-hM4D mice (hM4D - ; n = 7). g, h, Mechanical itch sensitivity ( g ) and spontaneous scratching ( h ) are unchanged following administration of CNO to aged NPY::Cre; Lbx1 FlpO ; R26 ds-hM4D mice (hM4D + ; n = 6) compared to aged control NPY::Cre; R26 ds-hM4D mice (hM4D - ; n = 6). i, j, Chemogenetic silencing of spinal NPY + neurons potentiates hyperknesis/alloknesis ( i ) and spontaneous scratching ( j ) in young but not aged mice treated with AEW to induce dry skin itch. k, Silencing spinal NPY + neurons exacerbates histamine alloknesis in young but not aged mice. Error bars represent SEM. *p < 0.05; **p < 0.01; ***p < 0.001; ns, no significant difference.
Rabbit α Gpr83, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 91/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rabbit+%CE%B1+gpr83/Anti-GPR83+Antibody/bio_rxiv__64898__2025__12__18__695289-158-35-38
Average 91 stars, based on 1 article reviews
rabbit α gpr83 - by Bioz Stars, 2026-09
91/100 stars
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human gpr83 cdna  (OriGene)
90
OriGene human gpr83 cdna
a, b, In aged mice, ablation of <t>Gpr83</t> + neurons by injection of PEN-SAP reverses mechanical hyperknesis ( a ; control blank SAP, n = 7; PEN-SAP, n =6) and prevents spontaneous scratching ( b ). c, d, Ablation of Tacr1 + neurons in aged mice by injection of SSP-SAP has no effect on mechanical itch sensitivity ( c ; SAP, n = 7; SSP-SAP, n =6) or spontaneous scratching ( d ). e, f, Mechanical itch sensitivity ( e ) and spontaneous scratching ( f ) are increased by administration of CNO to P56 NPY::Cre; Lbx1 FlpO ; R26 ds-hM4D mice (hM4D + ; n = 7) as compared to control NPY::Cre; R26 ds-hM4D mice (hM4D - ; n = 7). g, h, Mechanical itch sensitivity ( g ) and spontaneous scratching ( h ) are unchanged following administration of CNO to aged NPY::Cre; Lbx1 FlpO ; R26 ds-hM4D mice (hM4D + ; n = 6) compared to aged control NPY::Cre; R26 ds-hM4D mice (hM4D - ; n = 6). i, j, Chemogenetic silencing of spinal NPY + neurons potentiates hyperknesis/alloknesis ( i ) and spontaneous scratching ( j ) in young but not aged mice treated with AEW to induce dry skin itch. k, Silencing spinal NPY + neurons exacerbates histamine alloknesis in young but not aged mice. Error bars represent SEM. *p < 0.05; **p < 0.01; ***p < 0.001; ns, no significant difference.
Human Gpr83 Cdna, supplied by OriGene, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rabbit+%CE%B1+gpr83/GPR83+(NM_016540)+Human+Tagged+ORF+Clone/pm35559741-88-13-16
Average 90 stars, based on 1 article reviews
human gpr83 cdna - by Bioz Stars, 2026-09
90/100 stars
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rabbit polyclonal anti-gpr83  (GeneTex)
90
GeneTex rabbit polyclonal anti-gpr83
a, b, In aged mice, ablation of <t>Gpr83</t> + neurons by injection of PEN-SAP reverses mechanical hyperknesis ( a ; control blank SAP, n = 7; PEN-SAP, n =6) and prevents spontaneous scratching ( b ). c, d, Ablation of Tacr1 + neurons in aged mice by injection of SSP-SAP has no effect on mechanical itch sensitivity ( c ; SAP, n = 7; SSP-SAP, n =6) or spontaneous scratching ( d ). e, f, Mechanical itch sensitivity ( e ) and spontaneous scratching ( f ) are increased by administration of CNO to P56 NPY::Cre; Lbx1 FlpO ; R26 ds-hM4D mice (hM4D + ; n = 7) as compared to control NPY::Cre; R26 ds-hM4D mice (hM4D - ; n = 7). g, h, Mechanical itch sensitivity ( g ) and spontaneous scratching ( h ) are unchanged following administration of CNO to aged NPY::Cre; Lbx1 FlpO ; R26 ds-hM4D mice (hM4D + ; n = 6) compared to aged control NPY::Cre; R26 ds-hM4D mice (hM4D - ; n = 6). i, j, Chemogenetic silencing of spinal NPY + neurons potentiates hyperknesis/alloknesis ( i ) and spontaneous scratching ( j ) in young but not aged mice treated with AEW to induce dry skin itch. k, Silencing spinal NPY + neurons exacerbates histamine alloknesis in young but not aged mice. Error bars represent SEM. *p < 0.05; **p < 0.01; ***p < 0.001; ns, no significant difference.
Rabbit Polyclonal Anti Gpr83, supplied by GeneTex, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rabbit+%CE%B1+gpr83/gpr83+108229+antibody/pm36626872-227-27-25
Average 90 stars, based on 1 article reviews
rabbit polyclonal anti-gpr83 - by Bioz Stars, 2026-09
90/100 stars
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gpr83 antibody (oala00919)  (Aviva Systems)
N/A
GPR83 is an Orphan-A GPCR with an unknown ligand.
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rabbit anti-human gpr83 (extracellular domain)  (RayBiotech)
N/A
GPR83 Rabbit anti-Human Polyclonal (Extracellular Domain) (Unconjugated) Antibody, (50 µg)
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gpr83 rabbit polyclonal antibody  (OriGene)
N/A
Rabbit polyclonal anti GPR83 antibody
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gpr83 polyclonal antibody  (Elabscience)
N/A
GPR83 G Protein Coupled Receptor 83 is a Protein Coding gene Among its related pathways areSignaling by GPCRandPeptide ligand binding receptors Gene Ontology GO annotations related to this gene includeG protein coupled receptor activityandneuropeptide Y
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rabbit anti-human gpr83 polyclonal antibody  (Cusabio)
N/A
Rabbit anti-Human GPR83 Polyclonal Antibody
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anti-gpr83 monoclonal antibody  (Creative BioMart)
N/A
Orphan receptor. Could be a neuropeptide Y receptor.Store at -20°C or lower. Aliquot to avoid repeated freezing and thawing.http://www.creative-diagnostics.com/Anti-GPR83-MAb-173790-144.htm
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gpr83 antibody (oala00263)  (Aviva Systems)
N/A
GPR83 is an Orphan-A GPCR with an unknown ligand.
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rabbit anti-human gpr83 (c-terminus)  (RayBiotech)
N/A
GPR83 Rabbit anti-Human Polyclonal (C-Terminus) (Unconjugated) Antibody, (50 µg)
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Image Search Results


a, b, In aged mice, ablation of Gpr83 + neurons by injection of PEN-SAP reverses mechanical hyperknesis ( a ; control blank SAP, n = 7; PEN-SAP, n =6) and prevents spontaneous scratching ( b ). c, d, Ablation of Tacr1 + neurons in aged mice by injection of SSP-SAP has no effect on mechanical itch sensitivity ( c ; SAP, n = 7; SSP-SAP, n =6) or spontaneous scratching ( d ). e, f, Mechanical itch sensitivity ( e ) and spontaneous scratching ( f ) are increased by administration of CNO to P56 NPY::Cre; Lbx1 FlpO ; R26 ds-hM4D mice (hM4D + ; n = 7) as compared to control NPY::Cre; R26 ds-hM4D mice (hM4D - ; n = 7). g, h, Mechanical itch sensitivity ( g ) and spontaneous scratching ( h ) are unchanged following administration of CNO to aged NPY::Cre; Lbx1 FlpO ; R26 ds-hM4D mice (hM4D + ; n = 6) compared to aged control NPY::Cre; R26 ds-hM4D mice (hM4D - ; n = 6). i, j, Chemogenetic silencing of spinal NPY + neurons potentiates hyperknesis/alloknesis ( i ) and spontaneous scratching ( j ) in young but not aged mice treated with AEW to induce dry skin itch. k, Silencing spinal NPY + neurons exacerbates histamine alloknesis in young but not aged mice. Error bars represent SEM. *p < 0.05; **p < 0.01; ***p < 0.001; ns, no significant difference.

Journal: bioRxiv

Article Title: Age-related sensory dysfunction reconfigures the spinal circuitry for touch, itch and pain

doi: 10.64898/2025.12.18.695289

Figure Lengend Snippet: a, b, In aged mice, ablation of Gpr83 + neurons by injection of PEN-SAP reverses mechanical hyperknesis ( a ; control blank SAP, n = 7; PEN-SAP, n =6) and prevents spontaneous scratching ( b ). c, d, Ablation of Tacr1 + neurons in aged mice by injection of SSP-SAP has no effect on mechanical itch sensitivity ( c ; SAP, n = 7; SSP-SAP, n =6) or spontaneous scratching ( d ). e, f, Mechanical itch sensitivity ( e ) and spontaneous scratching ( f ) are increased by administration of CNO to P56 NPY::Cre; Lbx1 FlpO ; R26 ds-hM4D mice (hM4D + ; n = 7) as compared to control NPY::Cre; R26 ds-hM4D mice (hM4D - ; n = 7). g, h, Mechanical itch sensitivity ( g ) and spontaneous scratching ( h ) are unchanged following administration of CNO to aged NPY::Cre; Lbx1 FlpO ; R26 ds-hM4D mice (hM4D + ; n = 6) compared to aged control NPY::Cre; R26 ds-hM4D mice (hM4D - ; n = 6). i, j, Chemogenetic silencing of spinal NPY + neurons potentiates hyperknesis/alloknesis ( i ) and spontaneous scratching ( j ) in young but not aged mice treated with AEW to induce dry skin itch. k, Silencing spinal NPY + neurons exacerbates histamine alloknesis in young but not aged mice. Error bars represent SEM. *p < 0.05; **p < 0.01; ***p < 0.001; ns, no significant difference.

Article Snippet: The following primary antibodies were used in this study: rabbit α-Calcrl (1:50; Invitrogen), rabbit α-Calretinin (1:250; Swant), rabbit α-Caspase 3 (1:500; Invitrogen), rabbit α-DsRed (1:1000; Clontech), rabbit α-Galanin (1:500; Peninsula Lab), chicken α-GFP (1:000; Aves), rabbit α-Gpr83 (1:500; Alomone Labs), rat α-K8 (TROMA1, Developmental Studies Hybridoma Bank, 1:100), rabbit α-NF200 (1:1000; Sigma), mouse α-NF200 (1:500; Sigma), rabbit α-NK1R (1:500; Advanced Targeting Systems), rabbit α-NPY (1:500; Peninsula Lab), rabbit α-NPY1R (1:500; Neuromics), guinea pig α-prodynorphin (1:500; Abcam), rabbit α-Parvalbumin (1:1000; Swant), rabbit α-Pax2 (1:200; Zymed), rat α-RFP (1:1000; Chromotek), rabbit α-dsRed (1:1000 Clontech), guinea pig α-vGluT1 (1:1000; Millipore).

Techniques: Injection, Control

a, Representative images showing a lack of TUNEL staining in Y1-immunoreactive neurons in the dorsal horn at cervical segment C5 of young (left) and aged mice (right). b-d, Programmed cell death in populations of molecularly defined excitatory neurons identified by antibodies against Y1, Tacr1, Calcrl and Gpr83, and calretinin (CR) in aged compared to young mice: summaries of population size ( b ), the proportion of TUNEL + neurons expressing each molecular marker ( c ), and the proportion of each molecularly defined population labelled by TUNEL ( d ). e, Representative images showing a lack of TUNEL staining in Y1-immunoreactive neurons in the dorsal horn of control (left) and Merkel cell-silenced mice (right). f-h, Programmed cell death in populations of molecularly defined excitatory neurons identified by immunohistochemistry in Merkel cell-silenced mice compared to controls: summaries of population size ( f ), the proportion of TUNEL + neurons expressing each molecular marker ( g ), and the proportion of each molecularly defined population labelled by TUNEL ( h ). Scale bars, 20 µm. Error bars represent SEM. ns, no significant difference.

Journal: bioRxiv

Article Title: Age-related sensory dysfunction reconfigures the spinal circuitry for touch, itch and pain

doi: 10.64898/2025.12.18.695289

Figure Lengend Snippet: a, Representative images showing a lack of TUNEL staining in Y1-immunoreactive neurons in the dorsal horn at cervical segment C5 of young (left) and aged mice (right). b-d, Programmed cell death in populations of molecularly defined excitatory neurons identified by antibodies against Y1, Tacr1, Calcrl and Gpr83, and calretinin (CR) in aged compared to young mice: summaries of population size ( b ), the proportion of TUNEL + neurons expressing each molecular marker ( c ), and the proportion of each molecularly defined population labelled by TUNEL ( d ). e, Representative images showing a lack of TUNEL staining in Y1-immunoreactive neurons in the dorsal horn of control (left) and Merkel cell-silenced mice (right). f-h, Programmed cell death in populations of molecularly defined excitatory neurons identified by immunohistochemistry in Merkel cell-silenced mice compared to controls: summaries of population size ( f ), the proportion of TUNEL + neurons expressing each molecular marker ( g ), and the proportion of each molecularly defined population labelled by TUNEL ( h ). Scale bars, 20 µm. Error bars represent SEM. ns, no significant difference.

Article Snippet: The following primary antibodies were used in this study: rabbit α-Calcrl (1:50; Invitrogen), rabbit α-Calretinin (1:250; Swant), rabbit α-Caspase 3 (1:500; Invitrogen), rabbit α-DsRed (1:1000; Clontech), rabbit α-Galanin (1:500; Peninsula Lab), chicken α-GFP (1:000; Aves), rabbit α-Gpr83 (1:500; Alomone Labs), rat α-K8 (TROMA1, Developmental Studies Hybridoma Bank, 1:100), rabbit α-NF200 (1:1000; Sigma), mouse α-NF200 (1:500; Sigma), rabbit α-NK1R (1:500; Advanced Targeting Systems), rabbit α-NPY (1:500; Peninsula Lab), rabbit α-NPY1R (1:500; Neuromics), guinea pig α-prodynorphin (1:500; Abcam), rabbit α-Parvalbumin (1:1000; Swant), rabbit α-Pax2 (1:200; Zymed), rat α-RFP (1:1000; Chromotek), rabbit α-dsRed (1:1000 Clontech), guinea pig α-vGluT1 (1:1000; Millipore).

Techniques: TUNEL Assay, Staining, Expressing, Marker, Control, Immunohistochemistry